![B. Normal aldosterone stimulation: Activation of the angiotensin receptor (AT1R) by angiotensin II (AngII), or extracellular hyperkalemia, results in depolarization of the cell and resultant calcium influx via activated voltage-gated calcium channels. Calcium influx activates signaling to increase expression of aldosterone synthase and ultimately aldosterone production.](https://www.endotext.org/wp-content/uploads/2010/07/fig2b600.jpg)
B. Normal aldosterone stimulation: Activation of the angiotensin receptor (AT1R) by angiotensin II (AngII), or extracellular hyperkalemia, results in depolarization of the cell and resultant calcium influx via activated voltage-gated calcium channels. Calcium influx activates signaling to increase expression of aldosterone synthase and ultimately aldosterone production.